Tooth wear is a universal process. Every day, your teeth endure mechanical forces, chemical challenges, and frictional contact that slowly remove microscopic amounts of enamel. Over a lifetime, this is normal and expected. But when the rate of wear exceeds the body's capacity for repair — and enamel, critically, cannot regenerate — the distinction between different types of wear becomes clinically important. Enamel loss from erosion, abrasion, and attrition may look similar to the naked eye, but each has a distinct cause, pattern, and treatment approach.
Erosion is the chemical dissolution of enamel by acids that are not produced by oral bacteria. These acids come from two primary sources: dietary and intrinsic. Dietary acids include citrus fruits, carbonated beverages, sports drinks, wine, and vinegar-based foods. Even "healthy" choices like lemon water sipped throughout the day expose teeth to a continuous acid bath. Intrinsic acids come from within the body — most commonly from gastric reflux or frequent vomiting, as seen in gastroesophageal reflux disease and eating disorders.

The hallmark of erosion is a smooth, glazed appearance of the enamel surface. Unlike bacterial decay, which creates rough, cavitated lesions, erosion produces shallow, broad concavities known as cupping lesions, particularly on the chewing surfaces of molars. The enamel becomes progressively thinner, and the underlying yellow dentin begins to show through, giving teeth a darker, more translucent appearance.
Saliva is the body's natural defense against erosion. It buffers acids, supplies calcium and phosphate for remineralization, and forms a protective pellicle over enamel. Individuals with reduced salivary flow — due to medications, radiation therapy, or Sjogren's syndrome — are at substantially higher risk. A 2018 study in Caries Research found that 30 to 50 percent of adults show signs of erosive tooth wear, with the prevalence rising in younger populations due to increased consumption of acidic beverages.
Abrasion is the mechanical wear of tooth structure caused by objects or substances other than opposing teeth. The most common cause is overly aggressive toothbrushing, particularly with a hard-bristled brush and abrasive toothpaste. The characteristic pattern is a wedge-shaped notch, or V-shaped defect, at the gum line — most often on the buccal surfaces of canines and premolars, which protrude and receive the most brushing pressure.
Other sources of abrasion include improper use of interdental cleaning aids, habits like holding nails or pins between the teeth, and occupational exposure to abrasive dust in industries such as mining, sandblasting, and ceramics manufacturing. Pipe smoking also causes a distinctive notch on the teeth that hold the pipe stem.
Abrasion lesions are sharply defined with hard, smooth margins. Unlike erosion, which tends to be broad and shallow, abrasion produces deep, angular defects that can approach the pulp if left unchecked. The rate of progression depends on the frequency and force of the causative habit.
Attrition is the mechanical wear that results from contact between opposing teeth. Physiologic attrition occurs slowly throughout life as teeth rub together during chewing — an expected process that produces flattened cusps and shortened clinical crowns in older adults. Pathologic attrition, by contrast, is accelerated by bruxism, the habitual grinding or clenching of teeth, most often during sleep.
The pattern of attrition is diagnostic. Wear facets — flat, shiny, well-defined areas on the cusps of posterior teeth and the incisal edges of anterior teeth — match precisely between opposing arches. In severe bruxism, attrition can reduce the clinical crown height by several millimeters, exposing dentin and creating a characteristic "cupped out" appearance on the chewing surfaces.
Treatment depends entirely on the type of wear. Erosion requires identifying and eliminating the acid source, combined with remineralization strategies such as fluoride varnish and calcium phosphate pastes. Abrasion demands correction of the brushing technique and may require restoration of the defect with composite resin to prevent further progression. Attrition due to bruxism is managed with an occlusal splint, worn at night to separate the teeth and distribute forces.
Misdiagnosis leads to treatment failure. Filling an erosion lesion without addressing the underlying acid source guarantees the restoration will fail. Prescribing a night guard for abrasion misses the point entirely. The three mechanisms of tooth wear often coexist — a patient with bruxism who also drinks acidic beverages and brushes too hard develops a complex wear pattern requiring a comprehensive, multi-factorial approach. Correctly identifying the relative contribution of each is the foundation of effective treatment.
Jun 22
Jun 22

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